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11 , 12 Within cells, the GSH structure is relatively resilient because most intracellular proteases target -linked peptide bonds, while -linkages, such as those in this molecule, are generally unaffected

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whether these radical-trapping antioxidant pathways compensate for GPX4 downregulation in affected kidneys or if their dysfunction exacerbates ferroptosis susceptibility remains unknown, limiting multi-targeted therapeutic design