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Similar clinical symptoms are found in individuals with hereditary IF deficiency (also called congenital pernicious anemia) in whom the lack of IF results in the defective absorption of vitamin B 12

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and the absence of a transport system to export actively flavins in contrast to that of A

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In parallel, increased production of vasodilatory oxylipins may complement CIT-mediated improvements in NO bioavailability, collectively enhancing perfusion during and after exercise