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More importantly, Yesilova and colleagues (2005) documented that NAFLD/NASH patients exhibit the reduced activity of antioxidative mechanisms such as coenzyme Q10 and superoxide dismutase

Table 1 shows reagents that modulate ferroptosis in animal models or patients of these CNS diseases

Dynamic gene and protein expression patterns of the autism-associated met receptor tyrosine kinase in the developing mouse forebrain

This has resulted in hypo-cystinemia, with plasma cystine concentrations decreasing to 30% below baseline, as described in 12 patients with cirrhosis who received a FreeAmmine II source of amino acids in their intravenous hyperalimentation

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