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reported the therapeutic advantage of targeting SOD as a treatment for type-2 DN through the renal AMPK-PGC-1-Nrf2 and AMPK-FoxO signaling pathways (AMP-activated protein kinase-peroxisome proliferator-activated receptor coactivator 1 (PGC-1)-nuclear factor erythroid 2-related factor 2 and AMPK-forkhead box O transcriptional factors)
Perturbations of metabolic status within the pathway as ROS-processing activity increases could contribute to oxidant-triggered signalling
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This leads to a drop in pain-related signals traveling to the nervous system
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