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In 2025, an independent group at Brunel University London reported dose-dependent telomere length extension in normal human cells, the first solid Western replication of that claim

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The researchers commented that the peptide suppressed caspase-dependent apoptosis that increases during aging of cell cultures. By potentially restraining Caspase-3 activity, Epithalon might support cellular longevity and decrease apoptosis, enhancing its regenerative capabilities

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In yeast however, the proteolysis of H3 histone had been observed initially in sporulating and stationary phase cells, through the action of the vacuolar serine protease Prb1 [68]