The mechanism involves: Cholinergic antagonism -- cortistatin reduces cortical arousal by antagonizing acetylcholine-mediated excitation, effectively dampening the wake-promoting cholinergic system that maintains cortical desynchronization during wakefulness EEG synchronization -- the transition from wakefulness to SWS is characterized by increasingly synchronized, high-amplitude delta waves on EEG
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Mitra S, Behbahani H, Eriksdotter M
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Understanding the IGF-1 system in neurons, including the regulation of IGF-1 availability and signaling mechanisms, could be an important aspect for development of ALS therapeutic approaches 14
