Inflammation : High levels of inflammation in the brain are linked to increased blood-brain barrier (BBB) permeability [5] Stress : Acute stress activates brain mast cells that secrete proinflammatory cytokines [6] Bacterial infections : Bacteria in the brain increase MMP activity, which may induce the breakdown of the BBB [7] Toxins : LPS induces inflammation [8] Mold toxins : Mold toxins and mold components trigger inflammation and increase oxidative damage in the brain [9] High-fat , high-calorie diet : High-fat diets may increase oxidative damage, hypoxia, and inflammation in the brain [10] Leaky gut : Changes in the gut have been associated with BBB permeability [11, 12] Liver damage : In acute liver failure, the damaged liver releases MMP9 into the bloodstream, which may damage tight junctions [13] Diabetes or high blood sugar : Changes in glucose levels cause oxidative stress and inflammation [14] Disrupted sleep -wake cycle : Chronic sleep disturbance decreases glucose transport across the BBB and increases inflammation [15] Anything that triggers oxidative stress in glial cells and pericytes: Oxidative stress damages the BBB and causes structural and integrity problems [16, 17] Hypoxia : Hypoxia is a condition where there is not enough oxygen available
It is not necessarily ruined, but it indicates the formula is degrading
The 5mg dose offers 5 times the strength of the 1mg
Description N-Acetyl Selank Amidate Selank is a short, synthetic analogue of the tuftsin compound and as such mimics most of its effects
Each vial contains lyophilized Epithalon in a sealed 5ml glass vial, verified through Mile High Compounds three-tier quality protocol : 7x independent third-party analytical testing, 4-vial batch conformity verification, and comprehensive heavy metal screening
Participants lost an average of 1.8kg more than those taking a placebo over 12 weeks
