Rodent studies have investigated its role in stimulating new blood vessel formation (angiogenesis), accelerating wound tissue closure, and protecting the stomach and intestinal lining from experimentally induced damage
Authors concluded that, in the process of prostate carcinogenesis, activating histone modifications on GSTP1 are lost and subsequently DNA becomes methylated and inaccessible resulting in transcriptional silencing thus demonstrating interplay between histone reprogramming and promoter region methylation (Okino et al., 2007)
doi:10.1017/S0007680521000726
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