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The cellular and molecular changes triggered by ROS, as previously discussed, disrupt melanocyte homeostasis and contribute to pathological conditions 111

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BPC-157 appears to accelerate the formation of new microvessels by increasing VEGF expression in fibroblasts and endothelial cells, effectively fast-forwarding the vascularization phase of healing

This complex assessment could help to cluster patients into subgroups that can be better explained by specific biological dysfunctions