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In this case, GSH is functioning as a storage mechanism for cysteine which is otherwise unstable in its free form

doi: 10.1016/j.bbagen.2012.10.012 [DOI] [PubMed] [Google Scholar] 40.Galter D, Mihm S, Droge W

(5) However, treatment with buprenorphine should only be initiated in patients whom there is a low potential of precipitating withdrawal

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Homocysteine transport into systemic circulation enables a normal Hcy flux through the trans-sulfuration pathway, which would explain normal GSH levels in our cohort of vitamin B 12 deficient patients