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Notably, the diffusion of FITC-dextran was not observed, confirming that the excreted glucose was not the result of diffusion, but occurred via the glucose transporter (Supplementary Fig

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We further validated the ubiquitination of PPAR by NEDD4

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Phase Ib/II study of capmatinib (INC280) plus gefitinib after failure of epidermal growth factor receptor (EGFR) inhibitor therapy in patients with EGFR-mutated, MET factor-dysregulated nonsmall-cell lung cancer