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S2), in contrast to SIM-9 cells in which cholesterol enrichment did not modify mitochondrial GSH content (1.96 1.18 nmol/mg protein vs

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Overexpression of disintegrin and metalloproteinase domain-containing protein 9 (ADAM9) in lung cancer reduced LDLR protein stability by promoting its proprotein convertase subtilisin/kexin type 9 (PCSK9)-mediated degradation, leading to decreased cholesterol uptake and triggering cholesterol biosynthesis