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This leaves the developing brain vulnerable to oxidative stress and mitochondrial injury

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a reduced form of GSH dimerizes to its oxidized form, glutathione disulfide GSSG(Owen & Butterfield, 2010), such that the ratio of GSH to GSSG gives information about the cells healthy response to OS

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Furthermore, GGCT formed a tightly regulated positive feedback loop together with REST and miR-34a-5p

Reactive nitrogen species (RNS) such as nitric oxide promote damage to mitochondrial components, leading to dissipation of mitochondrial membrane potential and further increases in ROS and RNS production [42, 43]