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Variants in genes such as PNPLA3 , TM6SF2 , MBOAT7 , and GCKR have been linked to altered hepatic lipid metabolism, fibrosis susceptibility, and disease severity

It's the variable that can invalidate an entire research project

Westphal LM, Polan ML, Trant AS

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Then there is the Bcl-2-associated X protein(Bax) subfamily, which has pro-apoptotic activity and can form holes within the mitochondrial membrane (80), resulting in mitochondrial outer membrane permeabilization (MOMP) as well as a short discharge of cytochrome c (81, 82)