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The strain hydrolyzed the ester linkage at the C4 position to produce HT-2 toxin which is in turn hydrolyzed at the C15 ester linkage to produce T-2 triol

Although monophenolase activity was strongly impaired, the mutants still exhibited weak activity with the classical monophenolic substrates (tyramine and/or L -tyrosine)

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The pathogenesis of DN involves a variety of factors, such as altered haemodynamics, oxidative stress, and inflammation, and studies from animal models suggest that mitochondrial dysfunction plays an important role in the development of DN