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Drugs that promote healing and tissue regeneration often work by manipulating pathways like angiogenesis (blood vessel growth) and cell proliferation

However, these interventions are often used as adjunct therapies, and their clinical efficacy may be inconsistent due to individual differences and complex mechanisms

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There are few neurotransmitters, neuromodulators or growth factors capable of satisfying the above listed criteria and preventing AD-associated dysfunctions

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