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Animal As a senolytic, FOXO4-DRI carries mechanism-specific theoretical risks discussed in the senescence literature including disruption of beneficial senescence surveillance and off-target effects on stem-cell populations that share features with senescent cells and senolytic translation from mouse to human has a history of failures
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Importantly, genes such as Marco, involved in foam cell formation and matrix degradation[57, 58], Csf3, reported to be significantly increased in human AAA tissues[59, 60], Chil1, shown to be involved in vSMC phenotype switching [61, 62] and Olfm4, involved in pro-inflammatory response [63, 64] were uniquely downregulated in Mlkl / -d3-PPE animals
