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APLs are typically produced when the T cell receptor (TCR) contact sites are manipulated in immunogenic peptides
Cancer stem-like properties and gefitinib resistance are dependent on purine synthetic metabolism mediated by the mitochondrial enzyme MTHFD2
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Metabolic research also investigates MOTS-c, a mitochondrial-derived peptide that activates AMPK for energy homeostasis through complementary mitochondrial signaling mechanisms
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