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Substantial evidence suggests that the shed A-subunit (rather than the holoreceptor) is the autoantigen initiating and/or driving the autoimmune response to the TSHR in GD [for a recent review, see Ref

Gastroenterology 142 , 947956 (2012)

To protect itself, she went on, the body converts excess adrenaline into hydrogen peroxide which is then expelled through our pores and hair follicles

At 37C (body temperature or warm shipping conditions), that rate doubles

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Sustained AhR activity programs memory fate of early effector CD8+ T cells