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Concretely, lactylation of PDHA1 at K336 and CPT2 at K457/8 results in the inactivation of both enzymes and the inhibition of OXPHOS by restricting acetyl-CoA influx from pyruvate and fatty acid oxidation, respectively

In one study carried out on 2898 participants who lived in an area with factory-released dioxin, 425 diabetes (14.7%) subjects were identified, and a higher serum dioxin level is a risk factor for developing diabetes mellitus (Huang et al

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