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M.Romero-PuertasM

GHK-Cu suppressed the phosphorylation of p38 MAPK, JNK1/2 and NF-B p65 in LPS-induced ALI in mice The MAPK pathways have been demonstrated to participate in the activation of NF-kB in LPS-induced ALI

Research Applications Common Use Used in research to study repair, recovery, and protective cellular functions

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Future drug treatments should aim to address disease progression through neuroprotective strategies, such as gene therapy, immunotherapy targeting alpha-synuclein, and neurorestorative approaches that promote neurogenesis and synaptic plasticity